How Leptin and Insulin are Connected
Insulin levels increase within our body when we ingest any kind of food. Whenever we consume simple sugars or refined carbohydrates, insulin levels are significantly raised in prominent amounts much faster. Protein and complex carbohydrate content in your diet helps to ensure that insulin levels are raised in a more controlled and much slower manner.
Whenever we consume a large meal, our body responds by causing a large surplus of insulin to circulate. How much is released depends on quantity and more especially the food type. Excess simple sugars and carbs require an immediate high insulin response.
Blood sugar acts as our body’s fuel and insulin helps to deliver it to the cells in our body. For active individuals, this includes muscle cells situated within the body. Regardless of how active you are, your brain requires a steady amount of blood sugar to function in an optimum manner. After each meal, insulin has the task of transporting sugar throughout your system, seeking cells that require sugar in the body.
Our liver can receive approximately 60% of the sugar delivered by insulin, in healthy individuals. The liver acts similar to a storage facility for glycogen and the blood sugar that is not immediately utilized by the body is stored in the liver as glycogen for short-term use.
The Leptin Connection
Some of the insulin transports blood sugar to your stored fat or white adipose tissue. The fat cells absorb this blood sugar, which activates your metabolism. This process produces the leptin hormone. Leptin enters the bloodstream and travels to the brain. The more food we consume, the more insulin we produce and the more leptin we make.
Once the body has eaten enough, the leptin levels become higher. This higher leptin level signals the brain but additionally sends another signal to the pancreas that satiety has been achieved. This mechanism stops the beta cells in the pancreas from producing insulin.
Those who eat the correct amount of food to complement their level of physical activity experience more controlled blood sugar, since their insulin and leptin levels rise and fall accurately and in a balanced and level manner.
However, when insulin is competing with too much blood sugar and has no cells to deposit the sugar to, the insulin reacts by stimulating triglyceride production. These triglycerides are stored as fat and weight gain results. Once triglycerides elevate within the bloodstream, they interfere with leptin reaching your brain and signaling you are full.
Unfortunately, a cycle of overeating can occur since your brain is not receiving the full signal. This situation is known as leptin resistance. Triglyceride formation is further driven by insulin, making it more likely that weight gain will continue.
Exercise More and Eat Less
The old adage of “exercising more and eating less” will help get your hormones back to functioning correctly. You will burn stored fat, stimulate your metabolism and hunger mechanisms to become regulated and thus weight loss will ensue. Your insulin levels will also be working properly.
Videos you on Leptin Resistance
Leptin Regulates Metabolism And Appetite
It lets your brain know how much fat is in your body. As leptin levels rise, your appetite diminishes. As leptin levels fall, your appetite increases.
It regulates the rate of fat breakdown. As leptin levels rise, your metabolic rate increases. As leptin levels fall, your metabolism slows more information here
Leptin Reset Easy Start Guide by Jack Kruse
The Leptin Prescription is one of the structural pillars to living optimally. To make it easy for you to understand, to follow to digest, we’ve put together this Leptin Easy Start Guide to get you going…. read more here
Your weight gain is not a simple matter of overindulgence and indolence, but of survival and miscommunication. You are NOT a gluttonous sloth with a predisposition for stuffing your face all hours of the day while planted firmly on your backside. There is a perfectly good answer as to WHY you eat too much and move too little. It’s just that no one bothered to tell you until now…. read more here